To address the function of the CCL25/CCR9 axis in enhancing the antitumor CD8 T cell cytotoxic activity, we generated disease-specific survival (DSS) curves by comparing the effect of the combination of CCR9, CCL25, CD8A, and CD8B genes as a proxy for the infiltration of CD8+CCR9+ cells and production of CCL25 by the tumor. This evidence concerns the gene CD8A and neoplasm.