However, the current threshold for ApoB as a risk modifier in patients with myocardial infarction is relatively underdeveloped and requires further study (3).The understanding of ApoB and atherosclerosis formation needs to be further developed, although there is evidence that trapping of particles of ApoB in the arterial wall is the underlying cause of atherosclerosis (8, 10).In our study, ApoB was an independent risk factor for MACE after acute myocardial infarction and was a better predictor of residual risk. Here, APOB is linked to myocardial infarction.