For instance, inhibition of FADS2 has been shown to promote ferroptosis in tumor cells.[14, 15] Overexpression of FADS2 in mesenteric adipocytes has been demonstrated to suppress inflammatory responses.[16] Conversely, in some cases, the activation of FADS1/2 and PUFAs has been shown to exert cell‐damaging effects. The gene discussed is FADS1; the disease is neoplasm.