When analyzing GATA3‐low associated genes and pathways in the luminal subtypes separately, the luminal B subtype demonstrated enrichment of ‘Granulocyte chemotaxis’ (Figure 3D; supplementary material, Figure S8), NF‐ĸB transcription factor targets (supplementary material, Figure S6A), gene regulators in the NF‐ĸB family, and SPI1 was found in GATA3‐low/luminal B. TWIST2 and GLI1, described as associated with cancer stemness and EMT [52, 53], were the top ranked regulators in luminal A/GATA3‐low tumors (supplementary material, Figure S6B). This evidence concerns the gene GLI1 and cancer.