Another study, by Li et al. in 2023, investigated the role of di-n-butyl phthalate (DBP) in BCa tumorigenesis and demonstrated that it promotes cancer progression by downregulating JUND, Dual specificity protein phosphatase 3 (DUSP3), and ATPase H+ transporting V1 subunit C2 (ATP6V1C2), while upregulating FOSB and Ras homolog family member Q (RHOQ) [166]. This evidence concerns the gene ATP6V1C2 and cancer.