Guan et al. in 2020 reported that exosomic miR-95, derived from tumor-associated macrophages (TAMs), can act as an oncogene by suppressing targeting JUNB of PCa cells, thus promoting cell proliferation, invasion, and the expression of epithelial-to-mesenchymal transition (EMT) markers [98] (Figure 2). The gene discussed is JUNB; the disease is neoplasm.