IL-6 facilitates Th17 differentiation, chronic inflammation, and defective skin barrier function, while elevated IL-6 levels in AD skin are linked to bacterial colonization and dermatitis severity (Zhou et al., 2007; Son et al., 2014; Kim and Kim, 2019; Navarini, French, and Hofbauer, 2011). The gene discussed is IL6; the disease is dermatitis.