Given that the WT and Cxcr5−/− cells were collected from the same host msLN and that the transcriptional pathways regulating cell division were significantly inhibited in the Cxcr5−/− migratory cDC2 cells, we conclude that CXCR5 likely regulates both the recruitment and homeostasis of these migratory DC populations within the LN following infection. The gene discussed is MSLN; the disease is infection.