Ren [38] found that AA exerted anti-tumor effects by decreasing the phosphorylation levels of PI3K, Akt, mTOR, and other enzymes, and it was able to induce the growth cycle of cancer cells to stop at the G0/G1 phase, and the cell survival rate of ovarian cancer was reduced by 50%, and the number of apoptotic cancer cells was increased by 7 to 10-fold after treatment with AA (40 μg·mL−1), which fully demonstrated the antitumor potential of AA in ovarian cancer cells. This evidence concerns the gene AKT1 and neoplasm.