Dysregulation of critical signaling pathways, such as the Phosphatidylinositol 3-Kinase (PI3K)/AKT Serine/Threonine Kinase (AKT)/mTOR and Wnt/β-catenin pathways, further promotes uncontrolled tumor cell growth, survival, and metastasis [6]. This evidence concerns the gene AKT1 and neoplasm.