The pathways enriched in the NNV-susceptible family at all time-points were (1) immune system—cytosolic DNA-sensing, NOD-like and C-type lectin receptor signaling; (2) cellular processes—necroptosis; (3) genetic information processing—translation (ribosome); (4) viral infectious diseases (influenza A, COVID-19), and bacterial infectious diseases (Yersinia, Salmonella and pathogenic E. coli and Staphylococcus aureus infections). This evidence concerns the gene CLEC4D and staphylococcus aureus infection.