On the other hand, participants who did not require respiratory support and developed LC (n = 7) exhibited higher levels of IFN-α (Figure 3E, p = 0.033) and IFN-β (Figure 3F, p = 0.019) compared to NLC (n = 4) with the same clinical characteristics. The gene discussed is IFNB1; the disease is laryngotracheoesophageal cleft.