Our results might provide a significant support to other data from literature that revealed alterations in type I IFN, including IFN-α and IFN-β, among individuals with post-acute sequelae of COVID-19 (from children to adults) as compared to healthy controls and those recovered from an acute infection of SARS-CoV-2 without long COVID [14,27]. This evidence concerns the gene IFNA2 and COVID-19.