In particular with regard to the potential of ferroptosis and oxidative stress pathways in the pathogenesis of these disorders, Biamonte et al. reported a correlation between circulating iron status and hematological index indicators during the COVID-19 progression [144], and a low baseline level of a ferroptosis marker (SLC7A11) may indicate an increased risk for long COVID-19, and associated cardiovascular and neurological dysfunction [145,146,147]. Here, SLC7A11 is linked to COVID-19.