CXCL8 and endometriosis: These chemokines, specifically IL-8 and GRO alpha, along with epithelial cell-derived neutrophil-activating protein (ENA)-78, eotaxin, and interferon-inducible protein (IP)-10, may contribute to the pathogenesis of endometriosis by activating macrophages, promoting inflammatory reactions, facilitating the adhesion of endometriotic tissues within the peritoneal cavity, and enhancing angiogenesis, which is crucial for disease progression [37].