Comprehensive descriptions of ncRNAs in ALL previously described in the literature [83,84,85,86,87,88] have revealed how both pediatric and adult B-ALL rely on ncRNA-mediated dysregulation of core leukemia-driving pathways, including PI3K/AKT/mTOR, JAK/STAT, NF-κB, cell cycle, and apoptosis. The gene discussed is AKT1; the disease is precursor B-cell acute lymphoblastic leukemia.