Another lncRNA, MALAT1, is implicated in fibrosis: MALAT1 upregulation in hepatocytes and stellate cells can drive epithelial–mesenchymal transition and collagen gene expression—a finding consistent with the observation that MALAT1 levels are higher in fibrotic MASLD [123,141,142]. The gene discussed is MALAT1; the disease is metabolic dysfunction-associated steatotic liver disease.