NFAT5 and intracerebral hemorrhage: Duan et al. [67] found that BMSCs-EXOs rich in miR-146a-5p played a neuroprotective and improved function after intracerebral hemorrhage (ICH) by reducing neuronal apoptosis and down-regulating interleukin-1 receptor-associated kinase1 (IRAK1) and nuclear factor of activated T cells 5 (NFAT5) expression.