S. aureus skin infection augments IL-33 expression in the skin,101 IL-33 concentrations are elevated in lesional skin of patients with AD,102 superantigen-positive S. aureus causes more IL-33 release by human KCs compared with superantigen-negative S. aureus103 and IL-33 promotes food allergy.104 IL-33 is not a basophil attractant but enhances basophil migration to eotaxin.83 TSLP plays a critical role in the accumulation of basophils in MC903-treated skin independent of IL-3.105 TSLP played no detectable role in the basophil influx in dLNs of SEB-exposed skin. The gene discussed is CCL11; the disease is skin infection.