KCNJ2 and cardiovascular disorder: Next, we revealed that Kir2.1 function was restored in endothelial cells with reduced expression of the scavenger receptor CD36, an established FA-translocase affiliated with cardiovascular disease [23], and in the VAT artery endothelium of obese CD36 knockout mice, suggesting that VAT inhibits Kir2.1 through CD36 [19].