Taken together, our findings suggest that the IFN‐γ signaling pathway drives neutrophil secretion of endogenous BAFF, which in turn activates the differentiation of splenic GC B cells into autoantibody‐producing plasma cells (Figure 7Q, Supporting Information), supporting the presence of an immunoregulatory axis during MG acute exacerbation. Here, IFNG is linked to myasthenia gravis.