Notably, the increase in BAFF secretion by neutrophils is influenced by multiple factors, and IFN‐γ has been suggested as a potential upstream signaling molecule capable of inducing BAFF secretion.[26, 27] Moreover, our GSEA results revealed that the IFN‐γ response pathway was activated in blood neutrophils during acute MG exacerbation (Figure 5H, Supporting Information). Here, IFNG is linked to myasthenia gravis.