Zhou et al. demonstrated that in bleomycin-induced pulmonary-fibrosis models, miR-125b-5p specifically suppresses EMT in lung epithelial cells and attenuates disease severity; over-expression of miR-125b-5p lowers the pro-apoptotic gene BAK1, diminishes EMT proteins (N-cadherin, vimentin) and elevates the epithelial marker E-cadherin, thereby mitigating fibrosis (110). Here, VIM is linked to pulmonary fibrosis.