Moreover, Tong-Hong Wang et al. (13) found that short-term (24-h) exposure to PM2.5 could activate the EGFR pathway in lung cancer cells, while long-term (90-day) exposure promoted tumor progression through the activation of EGFR and AhR, by enhancing the TMPRSS2-IL18 pathway. This evidence concerns the gene EGFR and neoplasm.