GLP‐1 RAs, such as exenatide, have been shown to induce pancreatic duct gland expansion and exacerbate chronic pancreatitis, potentially accelerating the formation of dysplastic lesions, including pancreatic intraepithelial neoplasia (PanIN), by increasing ductal cell proliferation and promoting acinar‐to‐ductal metaplasia [5, 6]. Here, GCG is linked to chronic pancreatitis.