Conversely, the genes FTH1, SQSTM1, HSPA6, TSPYL2, PHLDA2, ITGAX, and DNAJA4 are expected to act as versatile protein regulators, potentially suppressing tumor cell genetic instability and promoting autophagy, apoptosis, and other forms of cell death [26–31]. The gene discussed is ITGAX; the disease is neoplasm.