While infection of moDCs with either HCMV variant enhanced the expression of TLR3, cGAS, and hSTING (Fig. S3B), the CMVIL-10-deficient strain elicited more robust transcription and secretion of type-I interferons (IFNA/B) (Fig. 4A, right part; Fig. 4B). This evidence concerns the gene TLR3 and infection.