RIPK3 and myocardial infarction: Notably, in mice subjected to acute myocardial infarction, receptor‐interacting serine/threonine‐protein kinase 3 (RIPK3), a key necroptotic mediator, triggers ER stress via a RIP3‐Ca2+‐calmodulin‐dependent protein kinase II (CAMKII) signaling axis [242].