GLIS1 and acute kidney injury: To investigate the roles of PRC2 and GLIS1 in regulating mRTEC dedifferentiation after AKI, H2O2‐treated mRTECs were treated with a PRC2 inhibitor (Embryonic Ectoderm Development 226, EED226) and subsequently transfected with sh‐NC or sh‐GLIS1.