The proinflammatory cytokines TNF-α, IL-1β, IL-6, and IL-17A, along with the chemokines CXCL9 and CXCL10, and the adhesion molecule VCAM-1, play critical roles in the pathogenesis of both MASLD and SAH. This evidence concerns the gene CXCL9 and metabolic dysfunction-associated steatotic liver disease.