Interleukin (IL)‐6 and tumor necrosis factor (TNF)‐α, which are known to be upregulated in peritonitis, may contribute to the induction of TG2 not only in fibroblasts but also in endothelial cells (Bhedi et al., 2020; Luo et al., 2016), potentially through the Janus kinase 2 (JAK2)/signal transducer and activator of transcription 3 (STAT3) or nuclear factor kappa‐light‐chain‐enhancer of activated B cells (NF‐κB) signaling pathway (Liu et al., 2024; Luo et al., 2016). Here, NFKB1 is linked to peritonitis.