TNFRSF1B and neoplasm: The gene signature of TNFR2+ Tregs was significantly enriched in profiles associated with stable and highly suppressive Treg phenotypes, including perinatal expanded Tregs [35], tissue‐resident Tregs [36], tumor‐infiltrating Tregs [37], and sepsis‐related TNFR2+ Tregs [38] (Figure S13).