For CHGA‐derived proteoforms, it is generally understood that circulating levels of CgA (CHGA19‐457) and pancreastin (CHGA272‐319) are elevated in T1D subjects [44], and that several CHGA derived peptides (including vasostatin‐1) are autoantigens for β‐cell‐destructive diabetogenic T‐cells [45, 46, 47]. The gene discussed is CGA; the disease is type 1 diabetes mellitus.