UGT1A1 and Gilbert syndrome: In fact, genotyping in the seven subjects with an elevation of indirect bilirubin of >3.0 mg/dL in the current study showed a polymorphism (UGT1A1*28) associated with benign indirect hyperbilirubinemia in Gilbert’s Syndrome (Rotger et al., 2005; Lankisch et al., 2006) in six of these subjects.