Interestingly, unlike in conditions characterized by low‐grade inflammation such as obesity and type 2 diabetes [37, 38, 39], PPARγ activation in our model did not lead to a reduction in classical pro‐inflammatory cytokines This may be explained by the absence of low‐grade inflammation following DOXO treatment in our experimental setting. The gene discussed is PPARG; the disease is type 2 diabetes mellitus.