This aligns with the results of Clement et al., who also noted that podocytes in both human and animal model MCD expressed ANGPTL-4 much more than podocytes in other kidney diseases such as membranous nephropathy, with unchanged ANGPTL-4 levels in focal segmental glomerulosclerosis (FSGS). This evidence concerns the gene ANGPTL4 and membranous glomerulonephritis.