However, to the authors' knowledge, the full translation of these findings has yet to be described, even though the interplay between CAMKs, AMPK and appetite‐regulating hormones (i.e., promoting ghrelin‐mediated food intake, and possibly reducing leptin‐mediated satiety) may be of specific interest to obesity and diabetes research [15]. This evidence concerns the gene GHRL and obesity due to melanocortin 4 receptor deficiency.