In addition, clinical autopsy finds that the expression level of KIF3 is down-regulated in the prefrontal cortex of SCZ patients after death, and Kif3b± mice show obvious schizophrenia traits such as social disturbance and postsynaptic defects in follow-up studies (Alsabban et al., 2020; Yoshihara et al., 2021). This evidence concerns the gene KIF3A and schizophrenia.