PARP1 and neurodegenerative disease: Despite the scarce data on the role of CYP B in neurodegenerative diseases, an earlier in-vitro study pointed to the ability of overexpressed CYP B to attenuate PARP cleavage, DNA disintegration, and ROS-induced neurotoxicity mediated by beta amyloid 25–35 model (Oh et al., 2011).