CBLN1 and metabolic dysfunction-associated steatotic liver disease: Apoptosisoccurred in parallel with hepatocellular proliferation; the strongestnegative association of these changes was found specifically withlonger chain SM and Cer molecules (40 and above), which is supportedin NAFLD. We found contradicting correlationsamong Cer and SM molecular species and apoptosis; namely, unsaturationwas not, but the number of hydroxyl groups was a differentiation factor.Those species without a hydroxyl group were positively correlated,while those having 1–2 or 3 were systematically negativelycorrelated to the histological change (Appendix Table_10).