Over 30 genetic variants have been strongly linked to ALS pathogenesis, with frequent mutations observed in superoxide dismutase 1 (SOD1), trans‐reactive DNA‐binding protein (TARDBP, encoding TDP‐43 protein), chromosome 9 open reading frame 72 (C9ORF72), and fusion sarcoma (FUS) [3]. This evidence concerns the gene SOD1 and amyotrophic lateral sclerosis.