Loss‐of‐function FLCN mutation results in Birt‐Hogg‐Dubé syndrome through hyperactivation of TFEB nuclear transloation, characterized by benign skin tumours.[2] However, the regulation of Rag‐Ragulator complex, which is responsible for mTOR activation, is regulated in the context of lysosomal damage is still unclear. Here, FLCN is linked to benign neoplasm of skin.