In silico docking, strong interactions of key phytochemicals, especially quercetin, luteolin, and β‐sitosterol, with NAFLD‐related targets (PPAR‐α, PPAR‐γ, AMPK, and SREBP‐1c), support the mechanistic basis for its efficacy. The gene discussed is SREBF1; the disease is metabolic dysfunction-associated steatotic liver disease.