For example, dysregulated expression of DNA methyltransferases (DNMT1, DNMT3A, and DNMT3B) in endometriotic lesions, along with differential DNA methylation patterns in genes involved in inflammatory pathways, steroid signaling, and apoptosis, have been linked to the development and progression of endometriosis[28,62]. This evidence concerns the gene DNMT3A and endometriosis.