JUN and myocardial infarction: Recent bioinformatic analyses have identified a set of ferroptosis- and autophagy-related genes, including Prostaglandin-Endoperoxide Synthase 2(PTGS2), Jun Proto-Oncogene, AP-1 Transcription Factor Subunit(JUN), NAD(P)H Quinone Dehydrogenase 1(NQO1), Nitric Oxide Synthase 3(NOS3), and Leptin Receptor(LEPR), as potential therapeutic targets in myocardial infarction and for reducing adverse cardiovascular events (58).