IFI44 and OASL have been associated with tumor progression and immune evasion, suggesting that they exert analogous roles in immune–tumor interactions.[83, 84, 85, 86] While TWF2 was the primary focus of this investigation, the remaining five genes demonstrate potential involvement in key tumor‐associated processes such as extracellular matrix remodeling, metabolic adaptation, and immune regulation. The gene discussed is OASL; the disease is neoplasm.