In the intestine, ST2 has been observed as less robustly expressed on ILC2s than at other barrier sites: ILC2‐conditional ST2 knockout mice in one study experienced partial loss of ILC2 numbers in the small intestine, less substantial than that observed in the lung, adipose tissue, or mLN [30], supporting that IL‐33 may contribute to immunity to Nb at the lung phase of infection. The gene discussed is IL33; the disease is infection.