No decrease in ILC2 IL‐13 production was noted, but in light of earlier discussed literature indicating ILC2s as potential enablers of TH2 functionality, IL‐33 likely plays a non‐redundant role in the expulsion of Nb, with contributions at the lung and intestinal stages of infection, and impacts on ILC2s and T cells still to be fully explored. The gene discussed is IL33; the disease is infection.