The current model of classical alarmin function in immunology, epithelium‐produced and APC detected, is becoming less supportable: IL‐25 being essentially a tuft cell product, IL‐33 being produced by many cell types within and across tissues, and TSLP being recently characterised as a myeloid and stromal cell product, and all best characterised as functionally signalling lymphoid effector cells as their primary contribution to various helminth infections, with only marginal redundancy between them (Table 1). Here, IL25 is linked to helminthiasis.