This finding is consistent with what is described by Argilés et al. (2014) and Fearon et al. (2012) on the pathophysiology of anorexia in the context of ACS, where the underlying mechanism is predominantly mediated by neuroendocrine and pro-inflammatory changes, such as increased pro-inflammatory cytokines (TNF-α, IL-6) and decreased ghrelin and other orexigenic stimuli—processes that are not dependent on chronological age [5,36]. Here, GHRL is linked to Anorexia.