A microarray study employing CasIIgly in HeLa cells (human cervical adenocarcinoma) revealed deregulation in mitochondrial function and perturbations in the phosphatidylinositol 3-kinase (PI3K)/protein kinase B (AKT)/mammalian target of rapamycin (mTOR) pathway (PI3K/AKT/mTOR pathway) [60], which is implicated in numerous cellular processes, for instance, proliferation, invasion, migration, apoptosis, glucose metabolism, and DNA repair, among others [60,61]. The gene discussed is MTOR; the disease is cervical adenocarcinoma.