Our results are further supported by Chaudhry et al. [39], who previously demonstrated increased frequencies of regulatory T cells (Tregs) and their activated subsets (HLA-DR− CD38+ and HLA-DR+ CD38+) in SAH patients, particularly during the delayed brain injury (DBI) phase and in those with post-aSAH complications such as cerebral vasospasm, seizures, and infections. The gene discussed is CD38; the disease is infection.