infection and present in the CM Ba, CM Bs, and CM Bm, might be involved in wound closure inhibition, we preincubated CM from infected cells and CM NI for 1 h with two concentrations of neutralizing antibodies against CXCL8 and CCL2 (0.5 and 1 μg/mL) before repeating the wound healing assay. This evidence concerns the gene CXCL8 and infection.