SLC2A1 and cancer: Biological evaluations, carried out across several cell lines, including gastric cancer (SGC7901), their glucose transport-deficient variant (SGC-7901R), HepG2, HeLa cells, and normal murine fibroblasts (L-929), showed significantly enhanced uptake of the complex in GLUT-overexpressing cancer cells compared with unmodified cisplatin, supporting the involvement of GLUTs in its internalization.