Conversely, we also identified molecules consistently reported across studies, reinforcing their established relevance to MM pathogenesis, such as the KIF gene and HLA-A, HLA-B and HLA-C antigens [52], which are involved in processes like cell proliferation, immune recognition and DNA repair, further supporting the link between genetic instability and tumorigenesis [52]. This evidence concerns the gene HLA-A and Miyoshi myopathy.